| 발표형식 : 구연
|
접수번호 - 470372 74 |
| Changes in AV Node Physiology Following Slow Pathway Modification |
| 媛��넧由�����븰援� �쓽怨쇰���븰 �닚�솚湲곕궡怨� |
| �씠留뚯쁺, �끂�깭�샇, �삤�슜�꽍, 吏꾩듅�썝, 諛뺤쿋�닔, 臾멸굔�썒, �쟾�몢�닔, �듅湲곕같, �솉�닚議�, 理쒓퇋蹂� |
Background: Dual AV node physiology often persists after radiofrequency catheter ablation(RFCA) for AV nodal reentrant tachycardia(AVNRT) but the mechanism of tachycardia eleimination is unresolved. Methods: AV node conduction characteristics were analyzed in 13 consecutive patients (Male 4, Age 50.8짹17.0) who showed dual AV node physiology after RFCA for AVNRT. We measured the changes of heart rate(HR), Wenckebach AV block cycle length(WAVBCL), effective refractory periods(ERP) of fast & slow pathway, maximum atrio-His intervals(Max. AH interval) of fast pathway & slow pathway before and after RFCA. Results: Each variable except ERP of fast pathway didn��셳 show any significant change between before and after RFCA(table). Conclusion: Even though we used the term of slow pathway modification, we couldn��셳 find any significant electrophysiologic changes of slow pathway followed slow pathway modification. The phenomenon of noninducibilty of AVNRT after slow pathway modification may reflect the alteration of properties of reentrant circuit resulted from the destruction of critical areas rather than the direct affection of the properties of the slow pathway.
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|
|
Pre-RFCA |
Post-RFCA |
P
value |
|
Heart
Rate(bpm) |
811.8짹154.6 |
802.2짹138.4 |
NS |
|
WAVBCL |
374.6짹35.7 |
376.9짹38.8 |
NS |
|
Fast Pathway
ERP |
359.2짹 54.5 |
320.0짹56.3 |
P=
0.02 |
|
Slow Pathway
ERP |
258.5짹41.2 |
253.9짹40.7 |
NS |
|
FP ERP - SP
ERP |
100.8짹38.0 |
73.9짹34.0 |
P=
0.01 |
|
Max. AH
interval(FP) |
196.8짹32.9 |
189.6짹38.0 |
NS |
|
Max. AH
interval(SP) |
344.2짹65.0 |
319.5짹47.0 |
NS |
|
Max. AH(SP)
– Max.
AH(FP) |
155.2짹58.4 |
129.9짹58.4 |
NS |
|
|
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